Metabolic Alterations associated with T2D are very well characterised by epidemiological and research primarily based experiments. The pathogenesis and development of T2D is ascribed to 4 mechanisms; amplified Sophisticated glycation stop solution (AGE) development, enhanced polyol pathway flux, activation of protein kinase C (PKC) isoforms, and improved hexosamine pathway flux[7]. https://www.directivepublications.org/journal-of-diabetology-research/